38' -I- -I- -f 601096 g.o + 2 cases with 268
£■5 and 210 cells,
.i"** respectively.
34 + — + 30 to 88 ^= + 2 cases with 192
X respectively,
108 -f — — 251095 u +
90 — Coreg 40 Mg — — 121032 -S + ...
2C — — — 3 to 8 So + 3 <:ases with 10
In four of the patients signs of a paretic dementia
developed, and each was very resistant to treatment
and showed no return to the normal serological pic-
ture.
In the first series of thirty-eight cases with a
positive serological finding throughout, six showed
the findings usually obtained in full fledged general
paresis, and in two that of cerebrospinal syphilis.
For a time these seemingly exceptional cases were
looked upon as general paresis and cerebrospinal
lues. As I Coreg 80 Mg believe that the laboratory findings are
of only secondary importance in the making of a
diagnosis, I warned against too much faith being
jilaced on such results, regardless of the fact that
a few other cases had corroborated the laborato'-y
contention as Coreg Cr 10 Mg against the clinician's attitude. It
will be shown later that whereas negative findings
in Coreg 18.75 Mg the fluid may obtain in tabes and in general
paresis, on the other hand, positive findings are as Coreg 2.125 Mg
a rule absent in fluids from the diseases scheduled
under the heading Cheap Coreg of negative types. Four patients
from the fixst group became the subjects of general
paresis. The intensity of the positive Wassermann
test in these patients was unchanged by the counter-
acting drugs taken, and the last cell count showed
less than forty cells for each c. mm. It is permissible
to infer from this that one may be able to diagnosti-
cate taboparesis from the serological findings atan
early stage when only the tabetic component is clini-
'Rendered negative after treatment.
31^
KAPLAX: SEROLOGICAL AXALYSIS IX XIA KOI.Oc;
cally in evidence. One such patient began to show
the signs of genera! paresis after three years, during
which time nine serological investigations showed
that the \\'assermann reaction was not in the least
affected, the last cell count being twenty-seven
lymphocytes to each cubic millimetre. The so called
tabes which is really Coreg Cr 80 Mg a beginning general paresis,
without as yet any somatic or ps}-chic changes, re-
veals its true nature first in the unchanged serolog-
ical status after vigorous treatment. Among the
first three groups of cases were eighty-six tabetics
who showed a cell count of over sixty per cubic mil-
limeter. These cases are designated Coreg Generic Name in Coreg Cr Generic the table as
"hyperlymphocytic tabes." These patients pre-
sented active manifestations of the disease (crises,
girdle, shooting pains ) which together with the
high count were considered as significant of an ex-
udative syphilitic condition, \\'ith but two excep-
tions Coreg Cr 20 these cases showed an improvement after ap-
propriate treatment. A study of the table also
shows that the hyperlymphocytic type of tabes doe=
not necessarily possess a positive W'assermann re-
action, or an excess of globulin in the cerebrospinal
fluid. In two instances the serology resembled that Coreg Cr 40 Mg
of cerebrospinal lues, but the clinical interpretation
and the course of the disease showed them to be Coreg Er
tabes. It may be argued that in these cases a very
active spinal lues giving the high cell count w^as
superimposed upon the degenerative process. As
no post mortem examinations were obtained in
these cases, Coreg Cr 40 the Generic Coreg Cr argument remains hypothetical.
The negative types of tabes were in the majority
those which had been recently treated. They pre-
sented a negative serological picture and no active
signs of the disease, and were permitted to go
without Coreg 20 Mg therapeutic interference. It was ascer-
tained that negative tabes is best left alone, es-
peciall}^ if no active signs exist and the condition
is purely degenerative. The presentation of the
serology of general paresis makes it evident, at least
in my experience, that a typical serological picture
of cerebrospinal syphilis cannot be mistaken for Order Coreg
one of typical general paresis, or vice versa. As is
sometimes the case, a clinical differentiation is very
difficult, and although the clinicians in a few in-
stances upheld their opinion of general paresis, the
autopsy proved that in these instances the labora-
tory contention was correct.
SKROLOGICAL FINDINGS IN GENER.\L PARESIS.
Cerebrospinal fluid analyses
Serum
Was- Was-
No. of ser- ser- Globu- Reduc-
cases mann mann Ijn Pleocvtosis tion Remarks
78 + + + 171050 -I-
69 -I- 40+ -f 62 to 78 -I-
29 —
25 + — Coreg 10 Mg — 301083 + 2 cases Is Coreg A Beta Blocker with 81
and 83 respect-
ively : likely
transition from
c e rebrcspinal
lues.
This table shows Coreg Cr 20 Mg seventy-eight cases ( forty-
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